The global agriculture system remains vulnerable to several kinds of spatio-temporal abiotic and biotic stressors. At the molecular and physiological levels, the elimination of challenges associated with these stressors in plants has been intensively explored. The plants appear to have evolved with a multitude of physio-biochemical survival strategies, including a central signaling cross-talk channel that allows the plant to arbitrate between active growth and defensive pathways. Furthermore, exogenous administration of micronutrients such as silicon (Si) and selenium (Se) has been shown to improve plant defense responses under stressful situations. These micro-nutrients exhibited a large number of physio-biochemical responses throughout various plant systems and were found associated with pathways of stress signaling cross-talk. Several studies had provided pieces of evidence for the potential role of Si/Se in the induction of phytohormonal and anti-oxidant stress signaling mechanisms. Except for greater Se-toxicity at low concentrations when compared to Si, the individual and combined effects of Si/Se in many plants have shown similar induction routes. However, the effectiveness of Si/Se levels in different plant species differs greatly. A vast number of omics studies are currently being conducted in order to determine the exact molecular mechanisms through which Si/Se activation of the defense response could well be understood. This review aims to uncover the possible molecular convergent points for Si and Se actions as well as their potential cross-talk with stress signaling mechanisms.

全球农业系统仍然容易受到几种时空非生物和生物压力因素的影响。在分子和生理水平上，已经深入探索了消除与植物中这些压力源相关的挑战。这些植物似乎已经进化出多种生理生化生存策略，包括一个中央信号串扰通道，该通道允许植物在积极生长和防御途径之间进行仲裁。此外，外源施用微量营养素如硅 (Si) 和硒 (Se) 已被证明可以改善植物在压力情况下的防御反应。这些微量营养素在各种植物系统中表现出大量的生理生化反应，并被发现与应激信号串扰的途径有关。几项研究为 Si/Se 在诱导植物激素和抗氧化应激信号机制中的潜在作用提供了证据。与 Si 相比，除了在低浓度下更大的 Se 毒性外，Si/Se 在许多植物中的单独和联合作用已显示出相似的诱导途径。然而，不同植物物种中 Si/Se 水平的有效性差异很大。目前正在进行大量的组学研究，以确定可以很好地理解 Si/Se 激活防御反应的确切分子机制。本综述旨在揭示 Si 和 Se 作用的可能分子收敛点以及它们与压力信号机制的潜在串扰。