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Prominin-2 Suppresses Ferroptosis Sensitivity.
Developmental Cell ( IF 10.7 ) Pub Date : 2019-12-02 , DOI: 10.1016/j.devcel.2019.11.004 Alexia Belavgeni 1 , Stefan R Bornstein 2 , Andreas Linkermann 3
Developmental Cell ( IF 10.7 ) Pub Date : 2019-12-02 , DOI: 10.1016/j.devcel.2019.11.004 Alexia Belavgeni 1 , Stefan R Bornstein 2 , Andreas Linkermann 3
Affiliation
Ferroptosis causes clinically relevant amounts of necrosis during the course of heart attacks and acute kidney injury. However, ferroptosis is still a very young research field. In this issue of Developmental Cell, Brown et al. describe the pentaspan membrane glycoprotein prominin-2 as a novel endogenous ferroptosis inhibitor.
中文翻译:
Prominin-2抑制Ferroptosis敏感性。
Ferroptosis在心脏病发作和急性肾损伤的过程中引起临床上大量的坏死。但是,铁锈病仍然是一个非常年轻的研究领域。在本期《发育细胞》中,Brown等人。描述了五萜类膜糖蛋白prominin-2作为一种新型内源性肥大症抑制剂。
更新日期:2019-12-02
中文翻译:
Prominin-2抑制Ferroptosis敏感性。
Ferroptosis在心脏病发作和急性肾损伤的过程中引起临床上大量的坏死。但是,铁锈病仍然是一个非常年轻的研究领域。在本期《发育细胞》中,Brown等人。描述了五萜类膜糖蛋白prominin-2作为一种新型内源性肥大症抑制剂。