Homolog of the yeast Fus3/Kss1 mitogen-activated protein kinase (MAPK) pathway and its target transcription factor, Ste12-like, are involved in penetration of host cuticle/pathogenicity in many ascomycete pathogens. However, details of their interaction during fungal infection, as well as their controlled other virulence-associated traits, are unclear.

中文翻译：

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Fus3/Kss1-MAP 激酶和 Ste12 样控制不同的生物防治特性，除了通过丝状真菌病原体中的磷酸化级联调节昆虫表皮渗透

酵母 Fus3/Kss1 丝裂原活化蛋白激酶 (MAPK) 通路的同系物及其靶转录因子 Ste12 样参与许多子囊菌病原体的宿主角质层渗透/致病性。然而，它们在真菌感染期间相互作用的细节，以及它们控制的其他毒力相关性状，尚不清楚。