ABSTRACT

Emerging adults with high levels of inhibited personality traits may be at-risk for drinking to cope during the COVID-19 pandemic. The current research explored mediational pathways between two inhibited personality traits (anxiety sensitivity (AS) and hopelessness (HOP)), internalizing symptoms (anxiety, depression, and COVID-19 distress), and coping drinking motives (drinking to cope with anxiety and drinking to cope with depression) during the pandemic. Cross-sectional data were collected from 879 undergraduate drinkers (79% female, 83% White, 18–25 years old) at five Canadian universities from January-April 2021. Participants self-reported on their personality, anxiety (GAD-7), depressive symptoms (PHQ-9), COVID-19 distress, and coping drinking motives. Mediational path analyses provided evidence of both specific and non-specific pathways between personality and coping motives via internalizing symptoms. Depressive symptoms partially mediated the link between HOP and drinking to cope with depression motives. While anxiety symptoms did not significantly mediate links between AS and coping with anxiety motives in the full model, evidence of mediation was found in a post-hoc sensitivity analysis. COVID-19 distress served as a non-specific mediator. AS and HOP are critical transdiagnostic risk factors that increase vulnerability for internalizing psychopathology and, in turn, risky drinking motives, in the context of the COVID-19 pandemic.

摘要

在 COVID-19 大流行期间，具有高度抑制人格特征的新兴成年人可能有饮酒以应对的风险。目前的研究探索了两种被抑制的人格特质（焦虑敏感性 (AS) 和绝望 (HOP)）、内化症状（焦虑、抑郁和 COVID-19 痛苦）和应对饮酒动机（饮酒以应对焦虑和饮酒）之间的中介途径以应对抑郁症）在大流行期间。横断面数据是从 2021 年 1 月至 4 月期间从加拿大五所大学的 879 名大学生饮酒者（79% 女性，83% 白人，18-25 岁）收集的。参与者自我报告了他们的性格、焦虑 (GAD-7)、抑郁症状 (PHQ-9)、COVID-19 困扰和应对饮酒动机。中介路径分析通过内化症状提供了人格与应对动机之间特定和非特定路径的证据。抑郁症状部分介导了 HOP 与饮酒之间的联系，以应对抑郁动机。虽然在完整模型中焦虑症状并未显着调节 AS 与应对焦虑动机之间的联系，但在事后敏感性分析中发现了调节的证据。COVID-19 困扰充当非特定调解人。AS 和 HOP 是关键的跨诊断风险因素，在 COVID-19 大流行的背景下，它们增加了内化精神病理学的脆弱性，进而增加了危险的饮酒动机。抑郁症状部分介导了 HOP 与饮酒之间的联系，以应对抑郁动机。虽然在完整模型中焦虑症状并未显着调节 AS 与应对焦虑动机之间的联系，但在事后敏感性分析中发现了调节的证据。COVID-19 困扰充当非特定调解人。AS 和 HOP 是关键的跨诊断风险因素，在 COVID-19 大流行的背景下，它们增加了内化精神病理学的脆弱性，进而增加了危险的饮酒动机。抑郁症状部分介导了 HOP 与饮酒之间的联系，以应对抑郁动机。虽然在完整模型中焦虑症状并未显着调节 AS 与应对焦虑动机之间的联系，但在事后敏感性分析中发现了调节的证据。COVID-19 困扰充当非特定调解人。AS 和 HOP 是关键的跨诊断风险因素，在 COVID-19 大流行的背景下，它们增加了内化精神病理学的脆弱性，进而增加了危险的饮酒动机。