Scurvy, caused by the prolonged deficiency of vitamin C (ascorbic acid), was once a malady predominantly associated with sailors in the 1600s and 1700s. However, the National Health and Nutrition Examination Survey in 2003-2004 found the overall prevalence of vitamin C deficiency in the United States to be 7.1%, suggesting that this is a more significant contributor to medical illness today than previously recognized.¹ While in the pediatric population the prevalence is <2% in 6- to 11-year-old children and <4% in adolescents, its frequency is increased in children/adolescents with autism spectrum disorder, many of whom manifest food selectivity and have restricted diets.¹ The relative rarity often leads to misdiagnosis or delayed diagnosis of scurvy and its complications. In this case, we report a unique presentation of scurvy in a 10-year-old boy with unilateral leg pain and bifrontal epidural hemorrhages.

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10 岁男孩继发于坏血病的双额硬膜外出血

由长期缺乏维生素 C（抗坏血酸）引起的坏血病曾是 1600 年代和 1700 年代主要与水手有关的疾病。然而，2003 年至 2004 年的全国健康和营养检查调查发现，美国维生素 C 缺乏症的总体患病率为 7.1%，表明这是导致当今医学疾病的一个比以前认识到的更重要的因素。¹虽然在儿科人群中，6 至 11 岁儿童的患病率 <2%，青少年的患病率 <4%，但患有自闭症谱系障碍的儿童/青少年的患病率增加，其中许多人表现出食物选择性并且有限制饮食。^1个相对罕见常常导致坏血病及其并发症的误诊或延误诊断。在本病例中，我们报告了一名 10 岁男孩的独特坏血病表现，伴有单侧腿痛和双额硬膜外出血。