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Biting into a union of oncology and metabolism through leukemic stem cells
Cell Metabolism ( IF 27.7 ) Pub Date : 2022-06-07 , DOI: 10.1016/j.cmet.2022.05.006 Mickie Bhatia 1 , Amro Elrafie 1
中文翻译:
通过白血病干细胞咬合肿瘤学和新陈代谢
更新日期:2022-06-07
Cell Metabolism ( IF 27.7 ) Pub Date : 2022-06-07 , DOI: 10.1016/j.cmet.2022.05.006 Mickie Bhatia 1 , Amro Elrafie 1
Affiliation
In this issue of Cell Metabolism, Liu et al. demonstrate that Prmt7 can regulate the onset and progression of leukemogenesis by inhibiting self-renewal capacity of leukemic stem cells (LSCs) as modeled in a murine version of chronic myeloid leukemia (CML).
中文翻译:
通过白血病干细胞咬合肿瘤学和新陈代谢
在本期《细胞代谢》中,Liu 等人。证明 Prmt7 可以通过抑制白血病干细胞 (LSC) 的自我更新能力来调节白血病发生的发生和进展,这在小鼠慢性髓性白血病 (CML) 中建模。