Pelvic fragility fractures result in significant morbidity and their incidence has increased over the past 30 years. One of the main risk factors in skeletal fragility is bone mineral density (BMD). Most of the current literature has focused on understanding spine and hip BMD. We aimed to measure the BMD of pelvis in a cohort of post-menopausal women and compare it to BMD at other skeletal sites. A questionnaire regarding risk factors for osteoporosis was completed by each participant. DXA scan of the pelvis was performed using research software. Three areas of the pelvis corresponding to common fractures were defined on pelvic DXA: R1 = symphysis public, R2 = inferior public rami, R3 = superior public rami. Pelvic BMD was calculated as the average BMD of R1-3. BMD at each location was reported as mean and standard deviation (SD). ANOVA was used to compare BMD between R1-R3 and pelvis, femoral neck, total hip, and spine. Pearson correlation was used to correlate pelvic BMD to BMD of proximal femur and spine. BMD was compared in four participant groups: 1- osteoporosis in spine and hip, 2- osteoporosis in spine only, 3-osteoporosis in hip only, and 4- no osteoporosis in spine and hip. The effect of diabetes and obesity on BMD at various skeletal sites was analyzed. Among the one hundred postmenopausal women enrolled in the study, age was: 64 ± 8, 31% were obese (BMI ≥ 30), and 8% had a diagnosis of type 2 diabetes. Pelvic area R3 had significantly higher BMD than R1 or R2 (p < 0.001). Pelvic BMD (0.50 ± 0.16) was significantly lower than total hip (0.70 ± 0.20) and spine BMD (0.97 ± 0.19) (p < 0.001). Pelvic BMD correlated with BMD at other skeletal locations, with the highest correlation with total hip (total hip: R2: 0.70, femoral neck R2: 0.50, spine R2: 0.65). Pelvic BMD was significantly lower in patients with osteoporosis of both hip and spine compared to the group without osteoporosis at both locations (p = 0.02). Obesity and type 2 diabetes were both associated with significantly higher BMD at pelvis, spine, and total hip. Pelvic BMD is lower than at other skeletal sites and is highly correlated with total hip area bone density. Obesity and type 2 diabetes are associated with higher pelvic BMD. To establish guidelines for the treatment pelvic BMD, studies defining the association of pelvic BMD with pelvic fracture risk are needed.

骨盆脆性骨折导致严重的发病率，并且其发病率在过去 30 年中有所增加。骨骼脆弱的主要风险因素之一是骨矿物质密度 (BMD)。目前的大多数文献都集中在了解脊柱和髋关节 BMD。我们旨在测量一组绝经后妇女的骨盆骨密度，并将其与其他骨骼部位的骨密度进行比较。每位参与者都完成了一份关于骨质疏松症危险因素的问卷调查。使用研究软件对骨盆进行 DXA 扫描。在骨盆 DXA 上定义了与常见骨折相对应的三个骨盆区域：R1 = 公共联合，R2 = 公共下支，R3 = 公共上支。骨盆 BMD 计算为 R1-3 的平均 BMD。每个位置的 BMD 报告为平均值和标准差 (SD)。ANOVA 用于比较 R1-R3 与骨盆、股骨颈、全髋和脊柱之间的 BMD。Pearson 相关性用于将骨盆 BMD 与股骨近端和脊柱。比较了四个参与者组的 BMD：1-脊柱和髋关节骨质疏松症，2-仅脊柱骨质疏松症，3-仅髋关节骨质疏松症，4-脊柱和髋关节无骨质疏松症。分析了糖尿病和肥胖对不同骨骼部位 BMD 的影响。在参加研究的 100 名绝经后女性中，年龄为：64 ± 8 岁，31% 肥胖（BMI ≥ 30），8% 被诊断为 2 型糖尿病。骨盆区域 R3 的 BMD 显着高于 R1 或 R2 ( p < 0.001)。骨盆 BMD (0.50 ± 0.16) 明显低于全髋 (0.70 ± 0.20) 和脊柱 BMD (0.97 ± 0.19) ( p< 0.001)。骨盆 BMD 与其他骨骼部位的 BMD 相关，与全髋相关性最高（全髋：R2：0.70，股骨颈 R2：0.50，脊柱 R2：0.65）。髋关节和脊柱骨质疏松患者的骨盆骨密度显着低于两个部位均未发生骨质疏松的患者（p  = 0.02）。肥胖和 2 型糖尿病均与骨盆、脊柱和全髋的 BMD 显着升高有关。骨盆 BMD 低于其他骨骼部位，并且与总髋部区域骨密度高度相关。肥胖和 2 型糖尿病与较高的骨盆 BMD 相关。为了建立治疗骨盆 BMD 的指南，需要研究确定骨盆 BMD 与骨盆骨折风险之间的关系。