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Correction: Surfaceome CRISPR screen identifies OLFML3 as a rhinovirus-inducible IFN antagonist
Genome Biology ( IF 10.1 ) Pub Date : 2021-11-15 , DOI: 10.1186/s13059-021-02534-5
Hong Mei 1 , Zhao Zha 1 , Wei Wang 1 , Yusang Xie 2 , Yuege Huang 1, 3 , Wenping Li 1, 3 , Dong Wei 4 , Xinxin Zhang 4 , Jieming Qu 2 , Jia Liu 1, 5, 6, 7, 8
Affiliation  

Correction to: Genome Biol 22, 297 (2021).

https://doi.org/10.1186/s13059-021-02513-w

Following publication of the original article [1], the authors identified an error in the author affiliations presented in additional file 1. The additional file has been updated and published in this correction.

The original article [1] has been corrected.

  1. 1.

    Mei H, Zha Z, Wang W, Xie Y, Huang Y, Li W, et al. Surfaceome CRISPR screen identifies OLFML3 as a rhinovirus-inducible IFN antagonist. Genome Biol. 2021;22(1):297. https://doi.org/10.1186/s13059-021-02513-w.

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Author notes
  1. Hong Mei, Zhao Zha and Wei Wang contributed equally to this work.

Affiliations

  1. Shanghai Institute for Advanced Immunochemical Studies and School of Life Science and Technology, ShanghaiTech University, Shanghai, 201210, People’s Republic of China

    Hong Mei, Zhao Zha, Wei Wang, Yuege Huang, Wenping Li & Jia Liu

  2. Department of Respiratory and Critical Care Medicine, Ruijin Hospital and Institutes of Respiratory Diseases, School of Medicine, Shanghai Jiao Tong University, Shanghai, 200025, China

    Yusang Xie & Jieming Qu

  3. University of Chinese Academy of Sciences, Beijing, 100049, China

    Yuege Huang & Wenping Li

  4. Research Laboratory of Clinical Virology, Ruijin Hospital, Shanghai Jiaotong University School of Medicine, Shanghai, 200025, China

    Dong Wei & Xinxin Zhang

  5. Shanghai Clinical Research and Trial Center, Shanghai, 201210, People’s Republic of China

    Jia Liu

  6. State Key Laboratory of Respiratory Disease, Guangzhou Medical University, Guangzhou, 510182, Guangdong Province, China

    Jia Liu

  7. Gene Editing Center, School of Life Science and Technology, ShanghaiTech University, Shanghai, 201210, People’s Republic of China

    Jia Liu

  8. Guangzhou Laboratory, No. 9 XingDaoHuanBei Road, Guangzhou International Bio Island, Guangzhou, 510005, Guangdong Province, China

    Jia Liu

Authors
  1. Hong MeiView author publications

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  2. Zhao ZhaView author publications

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  3. Wei WangView author publications

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  4. Yusang XieView author publications

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  5. Yuege HuangView author publications

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  6. Wenping LiView author publications

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  7. Dong WeiView author publications

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  8. Xinxin ZhangView author publications

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  9. Jieming QuView author publications

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  10. Jia LiuView author publications

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Corresponding authors

Correspondence to Jieming Qu or Jia Liu.

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Additional file 1: Fig. S1.

Construction of CRISPR genome-wide and surfaceome libraries. Fig. S2. Quality analyses of constructed genome-wide and surfaceome CRISPR libraries. Fig. S3. Evaluation of surfaceome and genome-wide CRISPR libraries. Fig. S4. Validation of the screening results. Fig. S5. Determination of gene modification efficiency. Fig. S6. Validation of the top 10 hits from surfaceome and genome-wide screens. Fig. S7. Construction and validation of single clones of ICAM-1−/−, RAB5C−/−, OLFML3−/−, SLC4A7−/− and ATP6AP1−/+ H1-Hela cells. Fig. S8. Validation of the effects of ICAM-1, RAB5C and OLFML3 on RV infection, related to Fig. 3. Fig. S9. Dissection of the functions of RAB5C and OLFML3 in RV infection. Fig. S10. RNA-seq analyses of the effects of RAB5C knockout on RV infection. Fig. S11. RNA-Seq analyses of the effects of OLFML3 on RV infection (related to Fig. 4). Fig. S12. Bar plots showing RT-qPCR quantification of ISG expression in mock and OLML3−/− cells at 24 h post infection of RV-B14 (a) and RV-A16 (b) at an MOI of 2

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Mei, H., Zha, Z., Wang, W. et al. Correction: Surfaceome CRISPR screen identifies OLFML3 as a rhinovirus-inducible IFN antagonist. Genome Biol 22, 314 (2021). https://doi.org/10.1186/s13059-021-02534-5

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  • DOI: https://doi.org/10.1186/s13059-021-02534-5

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更新日期:2021-11-16
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