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Hyperbaric oxygen therapy improves neurological function via the p38-MAPK/CCL2 signaling pathway following traumatic brain injury.
Neuroreport ( IF 1.6 ) Pub Date : 2021-09-06 , DOI: 10.1097/wnr.0000000000001719
Yingzi Jiang 1, 2 , Yuwen Chen 1, 2 , Chunling Huang 1, 2 , Anqi Xia 1, 2 , Guohua Wang 3 , Su Liu 1
Affiliation  

The anti-inflammatory mechanisms of hyperbaric oxygenation (HBO) treatment on traumatic brain injury (TBI)-induced neuroinflammation remain unclear. The aim of this study was expected the effect of HBO on CCL2-related signaling pathway following severe TBI in rats.

中文翻译:

高压氧治疗通过 p38-MAPK/CCL2 信号通路改善创伤性脑损伤后的神经功能。

高压氧(HBO)治疗创伤性脑损伤(TBI)引起的神经炎症的抗炎机制仍不清楚。本研究的目的是观察 HBO 对大鼠严重 TBI 后 CCL2 相关信号通路的影响。
更新日期:2021-09-06
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