Lesions in facet joints such as bone hyperplasia and degenerative changes in the intervertebral discs, can compress nerve roots and the spinal cord, leading to cervical spondylosis (CS). Lesions in these parts of the spine are commonly related to abnormal loads caused by bad posture of the cervical spine. This study aimed to understand the potential mechanical effects of load amplitude on cervical spine motion to provide a theoretical basis for the biomechanical causes of CS, and to provide a reference for preventing of the condition. In this study, a finite element model of the normal human cervical spine (C1-C7) was established and validated using an infrared motion capture system to analyze the effects of flexion angle on the stresses experienced by intervertebral discs, the anterior edge of the vertebral body, the pedicle, uncinate and facet joints. Our analysis indicated that the intervertebral disc load increased by at least 70% during the 20^∘ to 45^∘ flexion of the neck with 121% load increase in the vertebrae. In the intervertebral discs, the stress was largest at C4-C5, and the stress was moderate at C5-C6. These results are consistent with clinical CS prone site research. According to Wolff’s law, when bones are placed under large stresses, hyperplasia can result to allow adaptation to large loads. Increased cervical spine flexion angles caused the proliferation of bone in the above-mentioned parts of the spine and can accelerate accelerating the appearance of CS.

中文翻译：

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屈曲角度对颈椎病诱发机制影响的生物力学研究

小关节的病变，如骨质增生和椎间盘的退行性变化，会压迫神经根和脊髓，导致颈椎病（CS）。脊柱这些部位的病变通常与颈椎姿势不良引起的异常负荷有关。本研究旨在了解负荷幅度对颈椎运动的潜在力学影响，为研究CS的生物力学原因提供理论依据，并为该病的预防提供参考。在这项研究中，建立了正常人颈椎（C1-C7）的有限元模型，并使用红外运动捕捉系统进行了验证，以分析屈曲角度对椎间盘、椎体前缘受力的影响。身体，椎弓根，钩突和小关节。我们的分析表明，椎间盘负荷在 20 年间至少增加了 70%。^∘至 45 ^∘颈部屈曲，椎骨负荷增加 121%。椎间盘中C4-C5应力最大，C5-C6应力中等。这些结果与临床 CS 易发部位研究一致。根据沃尔夫定律，当骨骼承受很大的压力时，会导致增生以适应大负荷。颈椎屈曲角度增加导致上述脊柱部位骨质增生，可加速加速CS的出现。