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Inhibition of Long Noncoding RNA SNHG20 Improves Angiotensin II-Induced Cardiac Fibrosis and Hypertrophy by Regulating the MicroRNA 335/Galectin-3 Axis
Molecular and Cellular Biology ( IF 5.3 ) Pub Date : 2021-08-24 , DOI: 10.1128/mcb.00580-20 Mingyang Li 1 , Chunli Qi 1 , Renxing Song 1 , Chunming Xiong 1 , Xiao Zhong 1 , Ziguang Song 1 , Zhongping Ning 2 , Xiang Song 2
Molecular and Cellular Biology ( IF 5.3 ) Pub Date : 2021-08-24 , DOI: 10.1128/mcb.00580-20 Mingyang Li 1 , Chunli Qi 1 , Renxing Song 1 , Chunming Xiong 1 , Xiao Zhong 1 , Ziguang Song 1 , Zhongping Ning 2 , Xiang Song 2
Affiliation
Cardiac fibrosis is a hallmark of various heart diseases and ultimately leads to heart
failure. Although long noncoding RNA (lncRNA) SNHG20 has been reported to play important roles in various cancers, its function in cardiac
fibrosis remains unclear.
中文翻译：
抑制长链非编码 RNA SNHG20 通过调节 MicroRNA 335/Galectin-3 轴改善血管紧张素 II 诱导的心脏纤维化和肥大
心脏纤维化是各种心脏病的标志，最终导致心力衰竭。尽管据报道长链非编码 RNA (lncRNA) SNHG20 在各种癌症中发挥重要作用，但其在心脏纤维化中的作用仍不清楚。
更新日期：2021-09-12
中文翻译：
抑制长链非编码 RNA SNHG20 通过调节 MicroRNA 335/Galectin-3 轴改善血管紧张素 II 诱导的心脏纤维化和肥大
心脏纤维化是各种心脏病的标志，最终导致心力衰竭。尽管据报道长链非编码 RNA (lncRNA) SNHG20 在各种癌症中发挥重要作用，但其在心脏纤维化中的作用仍不清楚。
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