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BCAS3 exhibits oncogenic properties by promoting CRL4A-mediated ubiquitination of p53 in breast cancer
Cell Proliferation ( IF 5.9 ) Pub Date : 2021-07-09 , DOI: 10.1111/cpr.13088
Zhe Zhou 1 , Rongfang Qiu 1, 2 , Wei Liu 1 , Tianshu Yang 3 , Gen Li 3 , Wei Huang 3 , Xu Teng 3 , Yunkai Yang 4 , Hefen Yu 3 , Yang Yang 1, 2 , Yan Wang 1, 3, 4
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Breast cancer-amplified sequence 3 (BCAS3) was initially found to be amplified in human breast cancer (BRCA); however, there has been little consensus on the functions of BCAS3 in breast tumours.

中文翻译:

BCAS3通过促进CRL4A介导的p53泛素化在乳腺癌中表现出致癌特性

乳腺癌扩增序列3(BCAS3)最初被发现在人类乳腺癌(BRCA)中被扩增;然而,关于 BCAS3 在乳腺肿瘤中的功能几乎没有达成共识。
更新日期:2021-08-09
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