A new study shows that the SARS-CoV-2 nucleocapsid protein represses the antiviral type I interferon response through direct interaction with the signalling adaptor protein MAVS. Targeting this process might be a useful therapeutic strategy to boost immunity against COVID-19.

中文翻译：

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SARS-CoV-2 靶向 MAVS 进行免疫逃避

一项新研究表明，SARS-CoV-2 核衣壳蛋白通过与信号衔接蛋白 MAVS 的直接相互作用来抑制抗病毒 I 型干扰素反应。针对这一过程可能是提高对 COVID-19 免疫力的有用治疗策略。