Halomonas titanicae accelerated steel corrosion by dissimilatory Fe(III) reduction under anaerobic environments, and their adhesion was the key to achieving extracellular electron transfer between cells and Fe(III). This work investigated the inhibition effects of Cu and Ni alloying elements on corrosion of high strength low alloy (HSLA) steel affected by H. titanicae. It was found that both the addition of Cu (1.3%) and high content of Ni (7.2%) brought better corrosion resistance than the steel containing 4.8% Ni via decreasing the amount of sessile bacterial cells. And the inhibition efficiency of Cu with the lower content was stronger than that of Ni with the higher content. Biofilm inhibition mechanisms varied from Cu to Ni alloying elements, and the former was achieved via bactericidal Cu ions released from steel. While for the HSLA steel with high Ni content, the formation of nickel oxides including NiFe₂O₄ and Ni(OH)₂ refined the grains of corrosion products and decreased the bacterial attachment.

Halomonas Titanicae在厌氧环境下通过异化 Fe(III) 还原加速钢腐蚀，它们的粘附是实现细胞与 Fe(III) 之间细胞外电子转移的关键。这项工作研究了 Cu 和 Ni 合金元素对受H影响的高强度低合金 (HSLA) 钢腐蚀的抑制作用。泰坦尼克号. 结果表明，通过减少固着细菌细胞的数量，添加 Cu (1.3%) 和高含量 Ni (7.2%) 比含 4.8% Ni 的钢具有更好的耐腐蚀性。且Cu含量低的缓蚀效率强于Ni含量高的缓蚀效率。生物膜抑制机制从 Cu 到 Ni 合金元素各不相同，前者是通过从钢中释放的杀菌铜离子实现的。而对于高Ni含量的HSLA钢，NiFe ₂ O ₄和Ni(OH) ₂等镍氧化物的形成细化了腐蚀产物的晶粒并减少了细菌的附着。