Consciousness is considered a result of specific neuronal processes and mechanisms in the brain. Various suggested neuronal mechanisms have been laid forth, including the information integration theory, global neuronal work space, and neuronal construction of time and space as in the context of the temporo-spatial theory of consciousness. However, despite their focus on different neuronal mechanisms, these theories neglect the energetic-metabolic basis of the neuronal mechanisms that supposedly yield consciousness. Based on findings in physiological (sleep), pharmacological (general anesthesia) and pathological (vegetative state/unresponsive wakeful syndrome)-induced loss of consciousness in both human subjects and animals, we here suggest that energetic-metabolic processes focusing on adenosine-triphosphate, glucose, and γ-aminobutyrate/glutamate are indispensable for normal brain networks functional connectivity that renders consciousness possible. Therefore, we speak of energetic-metabolic predispositions of consciousness that complement current theories focused on the neural correlates of consciousness.

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书名页标题：能量如何支持我们的大脑产生意识：来自神经影像学的关于神经能量学假说的见解

意识被认为是大脑中特定的神经元过程和机制的结果。提出了各种建议的神经元机制，包括信息整合理论，全局神经元工作空间以及时空空间意识中上下文的时间和空间神经元构造。然而，尽管它们专注于不同的神经元机制，但这些理论忽略了据称产生意识的神经元机制的能量代谢基础。根据生理学（睡眠），药理学（全身麻醉）和病理学（植物人状态/无反应的清醒综合征）引起的人类和动物意识丧失的研究结果，我们在此提出以三磷酸腺苷为中心的能量代谢过程，葡萄糖，γ和氨基丁酸/谷氨酸对正常的大脑网络功能连接是必不可少的，从而使意识成为可能。因此，我们谈到了意识的能量代谢倾向，这些倾向补充了当前关注于意识的神经相关性的理论。