Induction of deactivation and apoptosis of hepatic stellate cells (HSCs) are principal therapeutic strategies for liver fibrosis. Krüppel-like factor 14 (KLF14) regulates various biological processes, however, roles, mechanisms and implications of KLF14 in liver fibrosis are unknown.

中文翻译：

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EZH2介导的KLF14表达抑制通过下调PPARγ促进HSC活化和肝纤维化

肝星状细胞 (HSC) 失活和凋亡的诱导是肝纤维化的主要治疗策略。Krüppel 样因子 14 (KLF14) 调节各种生物过程，然而，KLF14 在肝纤维化中的作用、机制和意义尚不清楚。