Blood cell aggregation in platelet-rich plasma (PRP) supplemented with the aggregation inducer ristomycin was studied following exposure to low-intensity extremely high frequency (EHF) radiation. Primary alcohols were used to stimulate partial degradation of the protein–lipid bilayer in the cell membrane. Exposure to EHF radiation was shown to decrease the extent of ristomycin-induced platelet aggregation and to change the slope of the aggregation curve. A biphasic character was observed for the effect of alcohols on platelet aggregation. The first phase was characterized by a decrease in aggregation and a decrease in the slope of the aggregation curve. In the second phase, platelet aggregation and the aggregation curve slope increased with the increasing alcohol concentration. A possible mechanism is discussed for the observed effects.

在暴露于低强度超高频（EHF）辐射后，研究了富含血小板诱导剂ristomycin的富血小板血浆（PRP）中的血细胞聚集。伯醇用于刺激细胞膜中蛋白质-脂质双层的部分降解。研究表明，暴露于EHF辐射下会降低弗瑞霉素诱导的血小板聚集程度，并改变聚集曲线的斜率。观察到醇对血小板聚集的影响具有两相特征。第一阶段的特征在于聚集的减少和聚集曲线的斜率的减少。在第二阶段，血小板聚集和聚集曲线斜率随着酒精浓度的增加而增加。为观察到的效果讨论了一种可能的机制。