### [Song et al. Page 1735][1] Altered receptor tyrosine kinase (RTK) activity has long been recognized as a key source of pro-mitotic and pro-survival signals that support the proliferation of cancer cells. The RTK EphA2 is particularly known to be overexpressed and to contribute to tumorigenic

中文翻译：

---

本期精选文章

### [Song等，Page 1735] [1]长期以来，公认的受体酪氨酸激酶（RTK）活性改变是支持癌细胞增殖的促有丝分裂和促存活信号的关键来源。众所周知，RTK EphA2过表达并有助于致瘤