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Disruption in the balance between apolipoprotein A‐I and mast cell chymase in chronic hypersensitivity pneumonitis
Immunity, Inflammation and Disease ( IF 3.1 ) Pub Date : 2020-10-04 , DOI: 10.1002/iid3.355 Yukihisa Inoue 1 , Tsukasa Okamoto 1 , Takayuki Honda 1 , Yoshihisa Nukui 1 , Takumi Akashi 2 , Tamiko Takemura 3 , Minoru Tozuka 4 , Yasunari Miyazaki 1
Immunity, Inflammation and Disease ( IF 3.1 ) Pub Date : 2020-10-04 , DOI: 10.1002/iid3.355 Yukihisa Inoue 1 , Tsukasa Okamoto 1 , Takayuki Honda 1 , Yoshihisa Nukui 1 , Takumi Akashi 2 , Tamiko Takemura 3 , Minoru Tozuka 4 , Yasunari Miyazaki 1
Affiliation
Apolipoprotein A‐I (apoA‐I) has an antifibrotic effect in idiopathic pulmonary fibrosis. Although pulmonary fibrosis is associated with poor prognosis of patients with hypersensitivity pneumonitis (HP), little is known regarding the role of apoA‐I in the pathogenesis of HP.
中文翻译:
慢性过敏性肺炎中载脂蛋白A-I和肥大细胞糜蛋白酶平衡的破坏
载脂蛋白 A-I (apoA-I) 在特发性肺纤维化中具有抗纤维化作用。尽管肺纤维化与过敏性肺炎 (HP) 患者的不良预后有关,但人们对 apoA-I 在 HP 发病机制中的作用知之甚少。
更新日期:2020-11-12
中文翻译:
慢性过敏性肺炎中载脂蛋白A-I和肥大细胞糜蛋白酶平衡的破坏
载脂蛋白 A-I (apoA-I) 在特发性肺纤维化中具有抗纤维化作用。尽管肺纤维化与过敏性肺炎 (HP) 患者的不良预后有关,但人们对 apoA-I 在 HP 发病机制中的作用知之甚少。