The Zonule of Zinn, or ciliary zonule, is the elaborate system of extracellular fibers that centers the lens in the eye. In humans, the fibers transmit forces that flatten the lens during the process of disaccommodation, thereby bringing distant objects into focus. Zonular fibers are composed almost entirely of 10–12 nm-wide microfibrils, of which polymerized fibrillin is the most abundant component. The thickest fibers have a fascicular organization, where hundreds or thousands of microfibrils are gathered into micrometer-wide bundles. Many such bundles are aggregated to form a fiber. Dozens of proteins comprise the zonule. Most are derived from cells of the non-pigmented ciliary epithelium in the pars plana region, although some are probably contributed by the lens and perhaps other tissues of the anterior segment. Zonular fibers are viscoelastic cables but their component microfibrils are rather stiff structures. Thus, the elastic properties of the fibers likely stem from lateral interactions between microfibrils. Rupture of zonular fibers and subsequent lens dislocation (ectopia lentis) can result from blunt force trauma or be a sequela of other eye diseases, notably exfoliation syndrome. Ectopia lentis is also a feature of syndromic conditions caused typically by mutations in microfibril-associated genes. The resulting ocular phenotypes raise the possibility that the zonule regulates lens size and shape, globe size, and even corneal topology, in addition to its well-recognized role in accommodation.

Zinn 小带或睫状小带是精细的细胞外纤维系统，将晶状体置于眼睛的中心。在人类中，纤维在不适应的过程中传递使晶状体变平的力，从而使远处的物体成为焦点。带状纤维几乎完全由 10-12 nm 宽的微原纤维组成，其中聚合原纤维蛋白是最丰富的成分。最粗的纤维具有束状组织，其中数百或数千个微纤维聚集成微米宽的束。许多这样的束聚集在一起形成纤维。数十种蛋白质构成小带。大多数来自平坦部区域的非色素睫状上皮细胞，尽管有些可能是由晶状体和前段的其他组织提供的。带状纤维是粘弹性电缆，但它们的组成微纤维是相当坚硬的结构。因此，纤维的弹性可能源于微纤维之间的横向相互作用。小带纤维断裂和随后的晶状体脱位（晶状体异位）可能是钝力外伤或其他眼部疾病的后遗症，尤其是剥脱综合征。晶状体异位也是典型由微纤维相关基因突变引起的综合征病症的特征。由此产生的眼表型提高了小带调节晶状体大小和形状、球体大小甚至角膜拓扑结构的可能性，除了它在调节中的公认作用。纤维的弹性可能源于微纤维之间的横向相互作用。小带纤维断裂和随后的晶状体脱位（晶状体异位）可能是钝力外伤或其他眼部疾病的后遗症，尤其是剥脱综合征。晶状体异位也是典型由微纤维相关基因突变引起的综合征病症的特征。由此产生的眼表型提高了小带调节晶状体大小和形状、球体大小甚至角膜拓扑结构的可能性，除了它在调节中的公认作用。纤维的弹性可能源于微纤维之间的横向相互作用。小带纤维断裂和随后的晶状体脱位（晶状体异位）可能是钝力外伤或其他眼部疾病的后遗症，尤其是剥脱综合征。晶状体异位也是典型由微纤维相关基因突变引起的综合征病症的特征。由此产生的眼表型提高了小带调节晶状体大小和形状、球体大小甚至角膜拓扑结构的可能性，除了它在调节中的公认作用。晶状体异位也是典型由微纤维相关基因突变引起的综合征病症的特征。由此产生的眼表型提高了小带调节晶状体大小和形状、球体大小甚至角膜拓扑结构的可能性，除了它在调节中的公认作用。晶状体异位也是典型由微纤维相关基因突变引起的综合征病症的特征。由此产生的眼表型提高了小带调节晶状体大小和形状、球体大小甚至角膜拓扑结构的可能性，除了它在调节中的公认作用。