Oreochromis niloticus was exposed to 10.0 ppb of organophosphate insecticide chlorpyrifos (CPF) and avermectin insecticides abamectin (ABM) and emamectin benzoate (EB) for 48 and 96 h. RBC and Hb decreased in CPF- and ABM-exposed fish after 96-h. Plasma ALT, AST, cortisol, and glucose increased in 96-h CPF-, ABM- and EB-exposed fish, while plasma ions declined in 96-h CPF-exposed ones. Insecticides caused alterations in liver oxidative stress parameters. In fish exposed to CPF, CAT increased after 48-h whereas it decreased after 96-h. Also, CAT declined in 48- and 96-h ABM-exposed fish, whereas it elevated in 48-h EB-exposed ones. Insecticides caused decreases in SOD at 48- and 96-h and in GR after 96-h. GSH elevated in CPF-exposed fish after 48-h, while it decreased in all the tested insecticide exposures after 96-h. Malondialdehyde of fish exposed to insecticides for 96-h increased. Consequently, toxic effects of insecticides on O. niloticus were generally as CPF > ABM > EB.

尼罗罗非鱼将其暴露于10.0 ppb的有机磷酸盐杀虫剂毒死（（CPF）和阿维菌素杀虫剂阿巴菌素（ABM）和阿马菌素苯甲酸酯（EB）中48和96小时。96小时后，暴露于CPF和ABM中的鱼的RBC和Hb降低。暴露于96 h CPF，ABM和EB的鱼血浆ALT，AST，皮质醇和葡萄糖增加，而暴露于96 h CPF的鱼血浆离子下降。杀虫剂引起肝脏氧化应激参数的改变。在暴露于CPF中的鱼中，CAT在48小时后增加，而在96小时后下降。同样，暴露于48 h和96 h暴露于ABM的鱼的CAT下降，而暴露于48 h EB暴露的鱼的CAT升高。杀虫剂导致48小时和96小时的SOD降低，96小时后的GR降低。暴露于CPF的鱼中的GSH在48小时后升高，而在所有测试的杀虫剂暴露后96小时后，GSH下降。暴露于杀虫剂96小时的鱼的丙二醛增加。因此，杀虫剂对尼罗罗非鱼通常为CPF> ABM> EB。