Figure 6A was incorrect in the original published paper. The images referring to the treatment with PMA or absinthin (Abs) 1 μM were mistakenly duplicated. The figure below now shows the correct image referring to the treatment with absinthin (Abs). The authors apologize for any inconvenience. Figure 6. Effect of absinthin (1) and derivatives on MUC5AC expression. BEAS-2B cells were incubated for 18 h with 10 nM PMA in the presence or absence of the indicated compounds: absinthin (1) (1 and 10 μM) and derivatives (1 μM; Ctrl, control untreated cell). (A) Immunofluorescence staining on treated cells (Ctrl; untreated control). MUC5AC in red, cytoskeleton stained with FITC-conjugated phalloidin (red) and nuclei stained with DAPI (blue). Magnification 200×. (B) Immunofluorescence quantification expressed as MUC5AC/actin fluorescence ratio. Results are means ± SEM of five fields in two independent experiments. (C) qRT-PCR analysis of MUC5AC gene expression. Results are expressed as 2-ΔΔCt and are means ± SEM of nine independent experiments. Significance levels: °P < 0.05 and °°P < 0.01 versus Ctrl; *P < 0.05 and **P < 0.01 versus PMA. This article has not yet been cited by other publications. Figure 6. Effect of absinthin (1) and derivatives on MUC5AC expression. BEAS-2B cells were incubated for 18 h with 10 nM PMA in the presence or absence of the indicated compounds: absinthin (1) (1 and 10 μM) and derivatives (1 μM; Ctrl, control untreated cell). (A) Immunofluorescence staining on treated cells (Ctrl; untreated control). MUC5AC in red, cytoskeleton stained with FITC-conjugated phalloidin (red) and nuclei stained with DAPI (blue). Magnification 200×. (B) Immunofluorescence quantification expressed as MUC5AC/actin fluorescence ratio. Results are means ± SEM of five fields in two independent experiments. (C) qRT-PCR analysis of MUC5AC gene expression. Results are expressed as 2-ΔΔCt and are means ± SEM of nine independent experiments. Significance levels: °P < 0.05 and °°P < 0.01 versus Ctrl; *P < 0.05 and **P < 0.01 versus PMA.

中文翻译：

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对人支气管上皮细胞中苦参素及其衍生物的抗炎活性的校正。

图6A在原始发表的论文中是不正确的。错误地复制了涉及使用1μMPMA或苦参碱（Absinthin）处理的图像。现在，下图显示了有关用苦参碱（Abs）治疗的正确图像。不便之处，敬请原谅。图6.苦参素（1）及其衍生物对MUC5AC表达的影响。在存在或不存在所示化合物的情况下，将BEAS-2B细胞与10 nM PMA孵育18小时：苦参碱（1）（1和10μM）及其衍生物（1μM; Ctrl，对照未处理的细胞）。（A）在处理的细胞上的免疫荧光染色（Ctrl；未处理的对照）。红色的MUC5AC，细胞骨架用FITC结合的鬼笔环肽（红色）染色，细胞核用DAPI染色（蓝色）。放大倍数200倍。（B）表示为MUC5AC /肌动蛋白荧光比的免疫荧光定量。结果是两次独立实验中五个场的平均值±SEM。（C）MUC5AC基因表达的qRT-PCR分析。结果表示为2-ΔΔCt，并且是九次独立实验的平均值±SEM。显着性水平：° P <0.05和°° P <0.01对Ctrl；* P <0.05和** P与PMA相比<0.01。本文尚未被其他出版物引用。图6.苦参素（1）及其衍生物对MUC5AC表达的影响。在存在或不存在所示化合物的情况下，将BEAS-2B细胞与10 nM PMA孵育18小时：苦参碱（1）（1和10μM）及其衍生物（1μM; Ctrl，对照未处理的细胞）。（A）在处理的细胞上的免疫荧光染色（Ctrl；未处理的对照）。红色的MUC5AC，细胞骨架用FITC偶联的鬼笔环肽（红色）染色，细胞核用DAPI染色（蓝色）。放大倍数200倍。（B）表示为MUC5AC /肌动蛋白荧光比的免疫荧光定量。结果是两次独立实验中五个场的平均值±SEM。（C）MUC5AC基因表达的qRT-PCR分析。结果表示为2-ΔΔCt，并且是九次独立实验的平均值±SEM。显着性水平：° P <0.05和°° P <0.01对Ctrl；* P <0.05和** P <0.01，相对于PMA。