Auditory verbal hallucinations (AVH) ― experienced as voice hearing independent of a corresponding external sound source ― are a cardinal symptom of psychosis. Approximately 6-13% of healthy individuals also experience voice hearing. Despite numerous attempts to explain the neurofunctional mechanisms underlying AVH, they remain notoriously unexplained. However, evidence relates AVH to mechanistic changes in the forward model. This review synthesizes behavioral and neuroimaging studies exploring the central role of cerebellar circuitry in the forward model, with a particular focus on non-verbal and verbal auditory feedback. It confirms that erratic prediction of sensory consequences in voice and sound production is linked to impaired cerebellar function, which initiates AVH and affects higher-level cognitive functions. We propose new research directions linking the forward model to voice and sound feedback processing. We consider this review as a starting point for mapping mechanisms of the forward model to neurocognitive mechanisms underlying AVH.

听觉幻觉（AVH）-经历了独立于相应外部声源的语音听觉-是精神病的主要症状。大约6-13％的健康个体也有声音听觉。尽管进行了大量尝试来解释AVH的神经功能机制，但众所周知，它们仍然无法解释。但是，有证据表明AVH与正向模型中的机械变化有关。这篇综述综合了行为和神经影像学研究，探讨了小脑回路在前向模型中的核心作用，尤其侧重于非语言和言语听觉反馈。它证实了对声音和声音产生中的感觉结果的错误预测与小脑功能受损有关，小脑功能受损会引发AVH并影响更高水平的认知功能。我们提出了新的研究方向，将前向模型链接到语音和声音反馈处理。我们认为本次审查是将正向模型的机制映射到AVH背后的神经认知机制的起点。