Among many Plasmodium proteins involved in the erythrocytic cycle, some exposed on the erythrocyte surface drive antigenic variability. Recently, Harrison et al. elucidated the structural basis by which RIFINs activate LILRB1 and suppress immune cell function. This breakthrough points to an additional strategy for survival in the human host.

中文翻译：

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RIFINing疟原虫-NK细胞相互作用。

在涉及红细胞周期的许多疟原虫蛋白质中，暴露在红细胞表面的一些疟原虫蛋白质驱动抗原变异性。最近，哈里森等人。阐明了RIFIN激活LILRB1并抑制免疫细胞功能的结构基础。这一突破表明了在人类宿主中生存的另一种策略。