Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus,Translational Neuroscience

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Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus
Translational Neuroscience ( IF 1.8 ) Pub Date : 2020-05-26 , DOI: 10.1515/tnsci-2020-0010
Chaohong Zhan _{1,

2,

3} , Gelei Xiao _{1,

2,

3} , Xiangyang Zhang _{1,

2,

3} , Xiaoyu Chen _{1,

2,

3} , Zhiping Zhang _{1,

2,

3} , Jingping Liu _{1,

2,

3}

Affiliation

Fibrosis in the ventricular system is closely associated with post-hemorrhagic hydrocephalus (PHH). It is characterized by an expansion of the cerebral ventricles due to CSF accumulation following intraventricular hemorrhage (IVH). The activation of transforming growth factor-β1 (TGF-β1) may be involved in thrombin-induced arachnoid fibrosis.

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