We present a new wave-type model of saltatory conduction in myelinated axons. Poor conductivity in the neuron cytosol limits electrical current signal velocity according to cable theory, to 1–3 m/s, whereas saltatory conduction occurs with a velocity of 100–300 m/s. We propose a wave-type mechanism for saltatory conduction in the form of the kinetics of an ionic plasmon-polariton being the hybrid of the electro-magnetic wave and of the synchronized ionic plasma oscillations in myelinated segments along an axon. The model agrees with observations and allows for description of the regulatory role of myelin. It explains also the mechanism of conduction deficiency in demyelination syndromes such as multiple sclerosis. The recently observed micro-saltatory conduction in ultrathin unmyelinated C fibers with periodic ion gate clusters is also explained.

我们提出了一种新的波型髓鞘轴突传导的模型。根据电缆理论，神经元细胞质中的导电性差将电流信号速度限制为1-3 m / s，而盐碱化传导则以100-300 m / s的速度发生。我们提出了一种盐类传导的波型机制，其形式为离子等离振子-极化子的动力学，它是电磁波和沿轴突的髓鞘部分中同步离子等离子体振荡的混合体。该模型与观察结果一致，并允许描述髓磷脂的调节作用。它还解释了脱髓鞘综合征（如多发性硬化症）中传导不足的机制。还解释了最近观察到的具有周期性离子门簇的超薄未髓鞘C纤维中的微盐化传导。