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Two classic mutations in the linker-helix IIL45 and segment IIS6 of Apolygus lucorum sodium channel confer pyrethroid resistance.
Pest Management Science ( IF 4.1 ) Pub Date : 2020-06-07 , DOI: 10.1002/ps.5944
Likui Wang 1 , Lianyun Lin 2 , Hao Wang 1 , Wenbo Duan 1 , Fen Li 1 , Kun Zhang 1, 2 , Peng Cao 3 , Zhiguang Yuchi 2 , Shaoying Wu 1
Affiliation  

Pyrethroids are classified as type I and type II for distinct symptomology. Voltage‐gated sodium channel is a primary target of pyrethroids. Mutations of the insect sodium channel have been identified to result in resistance to pyrethroids. Double mutation (L1002F/M906I) was detected in field‐strain of Apolygus lucorum (Meyer‐Dür). Although, it was illuminated the function of the same position mutation in other pests, it is necessary to demonstrate the role in A. lucorum .

中文翻译:

Apolygus lucorum钠通道的接头螺旋IIL45和IIS6区段中的两个经典突变赋予拟除虫菊酯抗性。

拟除虫菊酯因症状不同而分为I型和II型。电压门控钠通道是拟除虫菊酯的主要目标。已经发现昆虫钠通道的突变导致对拟除虫菊酯的抗性。在芦poly(Meyer-Dür)的田间菌株中检测到双突变(L 1002 F / M 906 I )。尽管它阐明了其他害虫中相同位置突变的功能,但有必要证明其在光合农杆菌中的作用。
更新日期:2020-06-07
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