Muscle disorders are characterized by differential involvement of various muscle groups. Among these, weakness predominantly affecting finger flexors is an uncommon pattern, most frequently found in sporadic inclusion‐body myositis. This finding is particularly significant when the full range of histopathological findings of inclusion‐body myositis is not found on muscle biopsy. Prominent finger flexor weakness, however, is also observed in other myopathies. It occurs commonly in myotonic dystrophy types 1 and 2. In addition, individual reports and small case series have documented finger flexor weakness in sarcoid and amyloid myopathy, and in inherited myopathies caused by ACTA1, CRYAB, DMD, DYSF, FLNC, GAA, GNE, HNRNPDL, LAMA2, MYH7, and VCP mutations. Therefore, the finding of finger flexor weakness requires consideration of clinical, myopathological, genetic, electrodiagnostic, and sometimes muscle imaging findings to establish a diagnosis.

中文翻译：

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手指屈肌无力的肌病：不仅包括包涵体肌炎。

肌肉疾病的特征在于不同肌肉群的参与程度不同。其中，主要影响手指屈肌的无力是一种罕见的现象，最常见于散发性包涵体肌炎。当在肌肉活检中未发现包涵体肌炎的全部组织病理学发现时，这一发现特别重要。然而，在其他肌病中也观察到明显的手指屈肌无力。它在强直性肌营养不良1型和2。另外，从个人报告和小病例系列通常发生已经记录在结节病和淀粉样肌病手指屈肌虚弱，和在引起遗传性肌病ACTA1，CRYAB，DMD，DYSF，FLNC，GAA，GNE，HNRNPDL，LAMA2，MYH7和VCP突变。因此，发现手指屈肌无力需要考虑临床，肌病理学，遗传，电诊断以及有时是肌肉成像的发现以建立诊断。