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Intestinal microbiota regulates tryptophan metabolism following oral infection with Toxoplasma gondii.
Parasite Immunology ( IF 1.4 ) Pub Date : 2020-04-10 , DOI: 10.1111/pim.12720 Liliane M Dos Santos 1 , Alessandra G Commodaro 2 , Alicia R R Vasquez 1 , Markus Kohlhoff 3 , Daniel A de Paula Guerra 3 , Roney S Coimbra 3 , Olindo A Martins-Filho 3 , Andrea Teixeira-Carvalho 3 , Luiz V Rizzo 4 , Leda Q Vieira 1 , Horacio M Serra 5, 6
Parasite Immunology ( IF 1.4 ) Pub Date : 2020-04-10 , DOI: 10.1111/pim.12720 Liliane M Dos Santos 1 , Alessandra G Commodaro 2 , Alicia R R Vasquez 1 , Markus Kohlhoff 3 , Daniel A de Paula Guerra 3 , Roney S Coimbra 3 , Olindo A Martins-Filho 3 , Andrea Teixeira-Carvalho 3 , Luiz V Rizzo 4 , Leda Q Vieira 1 , Horacio M Serra 5, 6
Affiliation
The intestinal microbiota plays an important role in modulating host immune responses. Oral Toxoplasma gondii infection can promote intestinal inflammation in certain mice strains. The IDO‐AhR axis may control tryptophan (Trp) metabolism constituting an important immune regulatory mechanism in inflammatory settings.
中文翻译：
弓形虫口腔感染后，肠道菌群调节色氨酸的代谢。
肠道菌群在调节宿主免疫反应中起重要作用。弓形虫的口腔感染可促进某些小鼠品系的肠道炎症。IDO-AhR轴可控制色氨酸（Trp）的代谢，从而构成炎症环境中的重要免疫调节机制。
更新日期：2020-04-10
中文翻译：
弓形虫口腔感染后，肠道菌群调节色氨酸的代谢。
肠道菌群在调节宿主免疫反应中起重要作用。弓形虫的口腔感染可促进某些小鼠品系的肠道炎症。IDO-AhR轴可控制色氨酸（Trp）的代谢，从而构成炎症环境中的重要免疫调节机制。
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