p75NTR−/− mice exhibit an alveolar bone loss phenotype and inhibited PI3K/Akt/β-catenin pathway,Cell Proliferation

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p75NTR−/− mice exhibit an alveolar bone loss phenotype and inhibited PI3K/Akt/β-catenin pathway
Cell Proliferation ( IF 5.9 ) Pub Date : 2020-03-25 , DOI: 10.1111/cpr.12800
Yingying Wang ₁ , Kun Yang ₂ , Gang Li ₁ , Rui Liu ₁ , Junyu Liu ₃ , Jun Li ₁ , Mengying Tang ₄ , Manzhu Zhao ₃ , Jinlin Song ₃ , Xiujie Wen _{1,

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Affiliation

The aim of this study was to investigate the role of p75 neurotrophin receptor (p75NTR) in regulating the mouse alveolar bone development and the mineralization potential of murine ectomesenchymal stem cells (EMSCs). Moreover, we tried to explore the underlying mechanisms associated with the PI3K/Akt/β‐catenin pathway.

中文翻译：

p75NTR−/− 小鼠表现出牙槽骨丢失表型并抑制 PI3K/Akt/β-catenin 通路

本研究的目的是探讨 p75 神经营养蛋白受体 (p75NTR) 在调节小鼠牙槽骨发育和小鼠外间充质干细胞 (EMSC) 矿化潜力中的作用。此外，我们试图探索与 PI3K/Akt/β-catenin 通路相关的潜在机制。

更新日期：2020-03-25

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