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Human monocytes subjected to ischaemia/reperfusion inhibit angiogenesis and wound healing in vitro
Cell Proliferation ( IF 5.9 ) Pub Date : 2020-01-19 , DOI: 10.1111/cpr.12753 Lars Hummitzsch 1 , Martin Albrecht 1 , Karina Zitta 1 , Katharina Hess 2 , Kerstin Parczany 1 , René Rusch 3 , Jochen Cremer 3 , Markus Steinfath 1 , Assad Haneya 3 , Fred Faendrich 4 , Rouven Berndt 3
Cell Proliferation ( IF 5.9 ) Pub Date : 2020-01-19 , DOI: 10.1111/cpr.12753 Lars Hummitzsch 1 , Martin Albrecht 1 , Karina Zitta 1 , Katharina Hess 2 , Kerstin Parczany 1 , René Rusch 3 , Jochen Cremer 3 , Markus Steinfath 1 , Assad Haneya 3 , Fred Faendrich 4 , Rouven Berndt 3
Affiliation
The sequence of initial tissue ischaemia and consecutive blood flow restoration leads to ischaemia/reperfusion (I/R) injury, which is typically characterized by a specific inflammatory response. Migrating monocytes seem to mediate the immune response in ischaemic tissues and influence detrimental as well as regenerative effects during I/R injury.
中文翻译:
体外缺血/再灌注的人单核细胞抑制血管生成和伤口愈合
初始组织缺血和连续血流恢复的顺序导致缺血/再灌注 (I/R) 损伤,其通常以特定的炎症反应为特征。迁移的单核细胞似乎介导缺血组织中的免疫反应,并影响 I/R 损伤期间的有害和再生作用。
更新日期:2020-01-19
中文翻译:
体外缺血/再灌注的人单核细胞抑制血管生成和伤口愈合
初始组织缺血和连续血流恢复的顺序导致缺血/再灌注 (I/R) 损伤,其通常以特定的炎症反应为特征。迁移的单核细胞似乎介导缺血组织中的免疫反应,并影响 I/R 损伤期间的有害和再生作用。