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Uric acid predicts long-term cardiovascular risk in type 2 diabetes but does not mediate the benefits of fenofibrate: The FIELD study.
Diabetes, Obesity and Metabolism ( IF 5.4 ) Pub Date : 2020-04-03 , DOI: 10.1111/dom.14046 Jacob Y Cao 1, 2 , Boris Waldman 1, 2 , Rachel O'Connell 1 , David R Sullivan 1, 2 , Russell S Scott 3 , Nanda Aryal 1 , Val Gebski 1 , Ian Marschner 1 , Marja-Riitta Taskinen 4 , John R Simes 1, 2 , Neil McGill 1, 2 , Alicia J Jenkins 1, 2 , Anthony C Keech 1, 2 ,
Diabetes, Obesity and Metabolism ( IF 5.4 ) Pub Date : 2020-04-03 , DOI: 10.1111/dom.14046 Jacob Y Cao 1, 2 , Boris Waldman 1, 2 , Rachel O'Connell 1 , David R Sullivan 1, 2 , Russell S Scott 3 , Nanda Aryal 1 , Val Gebski 1 , Ian Marschner 1 , Marja-Riitta Taskinen 4 , John R Simes 1, 2 , Neil McGill 1, 2 , Alicia J Jenkins 1, 2 , Anthony C Keech 1, 2 ,
Affiliation
To explore the relationship between baseline uric acid (UA) levels and long‐term cardiovascular events in adults with type 2 diabetes (T2D) and to determine whether the cardioprotective effects of fenofibrate are partly mediated through its UA‐lowering effects.
中文翻译:
尿酸可预测 2 型糖尿病患者的长期心血管风险,但不能调节非诺贝特的益处:FIELD 研究。
探讨基线尿酸 (UA) 水平与成人 2 型糖尿病 (T2D) 长期心血管事件之间的关系,并确定非诺贝特的心脏保护作用是否部分通过其 UA 降低作用介导。
更新日期:2020-04-03
中文翻译:
尿酸可预测 2 型糖尿病患者的长期心血管风险,但不能调节非诺贝特的益处:FIELD 研究。
探讨基线尿酸 (UA) 水平与成人 2 型糖尿病 (T2D) 长期心血管事件之间的关系,并确定非诺贝特的心脏保护作用是否部分通过其 UA 降低作用介导。