Heavy metals are one of the most hazardous pollutants in marine environments because of their bioaccumulation and biomagnification capabilities. Among them, cadmium (Cd) has been considered as one of the most dangerous for marine organisms. Here we incubated Ammonia cf. parkinsoniana specimens, a benthic foraminiferal taxon used in previous experiments, for up to 48 h in natural seawater with different concentrations of Cd to unravel the physiological change. We document a reduced pseudopodial activity of the Cd-treated specimens at concentrations >10–100 ppb in comparison with the control specimens. Moreover, confocal images of Cd-treated specimens using Nile Red as a fluorescent probe reveal an enhanced intracellular neutral lipid accumulation in the form of lipid droplets at 6 h and 12 h. This bioassay experiment allows for the direct evaluation of Cd-dose to A. cf. parkinsoniana-response relationships under laboratory controlled conditions and provides complementary information to field observations as well as to water quality guidelines and thresholds.

中文翻译：

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评估镉对底栖有孔虫氨的影响 参见。帕金森病：急性毒性试验

重金属是海洋环境中最危险的污染物之一，因为它们具有生物积累和生物放大能力。其中，镉（Cd）被认为是对海洋生物最危险的物质之一。在这里，我们孵化了氨 cf。parkinsoniana 标本，一种在以前的实验中使用的底栖有孔虫类群，在具有不同 Cd 浓度的天然海水中长达 48 小时，以解开生理变化。我们记录了与对照标本相比，浓度 >10-100 ppb 的镉处理标本的伪足活动降低。此外，使用尼罗红作为荧光探针的 Cd 处理标本的共聚焦图像显示，在 6 小时和 12 小时时，细胞内中性脂质以脂滴的形式积累增强。该生物测定实验允许直接评估 A. cf 的 Cd 剂量。实验室控制条件下的帕金森反应关系，并为现场观察以及水质准则和阈值提供补充信息。