Cancer-associated histone mutation H2BG53D disrupts DNA-histone octamer interaction and promotes oncogenic phenotypes.,Signal Transduction and Targeted Therapy

当前位置： X-MOL 学术 › Signal Transduct. Target Ther. › 论文详情

Our official English website, www.x-mol.net, welcomes your feedback! (Note: you will need to create a separate account there.)

Cancer-associated histone mutation H2BG53D disrupts DNA-histone octamer interaction and promotes oncogenic phenotypes.
Signal Transduction and Targeted Therapy ( IF 39.3 ) Pub Date : 2020-03-06 , DOI: 10.1038/s41392-020-0131-0
Yi Ching Esther Wan _{1,

2} , Tsz Chui Sophia Leung ₃ , Dongbo Ding ₃ , Xulun Sun ₃ , Jiaxian Liu _{1,

2} , Lina Zhu _{1,

2} , Tze Zhen Evangeline Kang _{1,

2} , Du Yang ₄ , Yuchen Zhang _{1,

2} , Jitian Zhang _{1,

2} , Chengmin Qian ₅ , Michael Shing Yan Huen ₅ , Qing Li ₄ , Maggie Zi Ying Chow ₆ , Zongli Zheng _{1,

2,

6} , Junhong Han ₇ , Ajay Goel ₈ , Xin Wang _{1,

2} , Toyotaka Ishibashi ₃ , Kui Ming Chan _{1,

2}

Affiliation