A laboratory experiment was performed to evaluate the potential role of H₂S on cadmium (Cd) toxicity in Phlox paniculata L. Seeds pretreated with 0.3, 0.6, 0.9, and 1.2 mM NaHS as a donor of H₂S for 24 h and subsequently exposed to 100, 200, and 300 μM CdCl₂ for 26 days had significantly higher germination rate compared with Cd alone. Meanwhile, 2-year-old seedlings sprayed with 0.3, 0.6, and 0.9 μM NaHS were grown in soil with 0.3, 0.6, and 1.2 mg/kg CdCl₂, respectively. We observed that H₂S decreased Cd accumulation in leaves and elevated Cd concentration in roots. Cd toxicity in seedlings resulted in a substantial increase in Cd-induced overproduction of malondialdehyde (MDA), Cd accumulation, and electrolyte leakage. Meanwhile, addition of NaHS increased photosynthetic performance compared with Cd alone. Exogenous H₂S significantly elevated biomass, improved antioxidant enzyme activities, and reduced ABA content compared with Cd alone. H₂S also plays an important role in the ABA signaling pathway during stress. Notably, NaHS promoted Cd uptake by Phlox paniculate L. from soil. The prediction model of H₂S for increasing plant resistance and reducing soil Cd pollution was established by factor analysis method based on comprehensive evaluation of plant stress physiology.

进行了一项实验室实验，以评估H ₂ S对福寿螺中镉（Cd）毒性的潜在作用。用0.3、0.6、0.9和1.2 mM NaHS作为H ₂ S供体预处理的种子持续24小时，随后与单独的Cd相比，暴露于100、200和300μMCdCl ₂中26天的种子发芽率明显更高。同时，分别在0.3、0.6和1.2 mg / kg CdCl _2的土壤中种植了喷有0.3、0.6和0.9μMNaHS的2岁树苗。我们观察到H ₂硫减少了叶片中镉的积累，而根部中的镉浓度却升高。幼苗中的Cd毒性导致Cd诱导的丙二醛（MDA）过量生产，Cd积累和电解质泄漏大量增加。同时，与单独使用Cd相比，添加NaHS可以提高光合性能。与单独的Cd相比，外源H ₂ S显着提高了生物量，提高了抗氧化酶的活性，并降低了ABA含量。在压力下，H ₂ S在ABA信号通路中也起着重要作用。值得注意的是，NaHS促进了福禄考从土壤中吸收镉。H ₂的预测模型在综合评价植物逆境生理的基础上，采用因子分析法建立了提高植物抗逆性和减少土壤镉污染的硫素。