Many papers evaluated the effect of the environmental, or occupational endocrine disruptors (ED), on the thyroid gland, that can lead to thyroid autoimmunity. A higher prevalence of autoimmune thyroid diseases (AITD) was observed in people living in polluted areas near to petrochemical plants, and in petrochemical workers, but also in area contaminated with organochlorine pesticides, or with polychlorinated biphenyls, or near aluminum foundries. The exposure to Hg in chloralkali workers, or in swordfish consumers has been also found to increase AITD prevalence. Vanadium has been shown to increase the inflammatory response of thyrocytes. A beneficial effect of omega-3 fatty acids, and of myo-inositol and selenomethionine have been shown to counteract the appearance of AITD in subjects exposed to environmental or occupational ED. More large studies are needed to investigate the potential roles of ED in the induction of AITD, and of agents or habits that are able to prevent them.

中文翻译：

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内分泌干​​扰物和甲状腺自身免疫。

许多论文评估了环境或职业内分泌干扰物（ED）对甲状腺的影响，该作用可导致甲状腺自身免疫。在石化厂附近污染地区，石化工人中，以及在有机氯农药，多氯联苯污染的地区或铝铸造厂附近，人们发现自身免疫性甲状腺疾病（AITD）的患病率较高。还发现氯碱工人或箭鱼消费者接触汞会增加AITD的患病率。钒已被证明可以增加甲状腺细胞的炎症反应。业已证明，omega-3脂肪酸以及肌醇和硒代蛋氨酸的有益作用可以抵消AITD在暴露于环境或职业性ED的受试者中的出现。