All-DNA scaffolds act as templates for the organization of photosystem I model systems. A series of DNA templates composed of ZnII -protoporphyrin IX (ZnII PPIX)-functionalized G-quadruplex conjugated to the 3'- or 5'-end of the tyrosinamide (TA) aptamer and ZnII PPIX/G-quadruplex linked to the 3'- and 5'-ends of the TA aptamer through a four-thymidine bridge. Effective photoinduced electron transfer (ET) from ZnII PPIX/G-quadruplex to bipyridinium-functionalized tyrosinamide, TA-MV2+ , bound to the TA aptamer units is demonstrated. The effectiveness of the primary ET quenching of ZnII PPIX/G-quadruplex by TA-MV2+ controls the efficiency of the generation of TA-MV+. . The photosystem-controlled formation of TA-MV+. by the different photosystems dictates the secondary activation of the ET cascade corresponding to the ferredoxin-NADP+ reductase (FNR)-catalysed reduction of NADP+ to NADPH by TA-MV+. , and the sequestered alcohol dehydrogenase catalysed reduction of acetophenone to 1-phenylethanol by NADPH.

中文翻译：

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用ZnII-原卟啉All-DNA G-四联体/适体支架模拟光合作用。

全DNA支架充当组织photosystem I模型系统的模板。一系列DNA模板由ZnII-原卟啉IX（ZnII PPIX）-官能化的G-四链体与酪氨酸酰胺（TA）适体的3'-或5'-末端缀合和ZnII PPIX / G-四链体连接到3'组成-和通过四胸苷桥的TA适体的5'端。证明了从ZnII PPIX / G-四链体到联吡啶官能化的酪氨酸酰胺TA-MV2 +的有效光诱导电子转移（ET），其与TA适体单元结合。TA-MV2 +对ZnII PPIX / G-四链体进行一次ET猝灭的有效性控制着TA-MV +生成的效率。。由光系统控制的TA-MV +的形成。不同光系统的作用决定了ET级联反应的二次激活，这对应于铁氧还蛋白-NADP +还原酶（FNR）催化的TA-MV +将NADP +还原为NADPH。，以及隔离的乙醇脱氢酶催化NADPH将苯乙酮还原为1-苯基乙醇。