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Impact of yeast and lactic acid bacteria on mastitis and milk microbiota composition of dairy cows.
AMB Express ( IF 3.5 ) Pub Date : 2020-01-29 , DOI: 10.1186/s13568-020-0953-8
Jing Gao 1 , Yu-Chen Liu 1 , Yu Wang 1 , Han Li 1 , Xiang-Ming Wang 1 , Yan Wu 1 , Ding-Ran Zhang 1 , Si Gao 1 , Zhi-Li Qi 1
Affiliation  

This experiment was conducted to evaluate the impact of yeast and lactic acid bacteria (LAB) on mastitis and milk microbiota composition of dairy cows. Thirty lactating Holstein cows with similar parity, days in milk were randomly assigned to five treatments, including: (1) Health cows with milk SCC < 500,000 cells/mL, no clinical signs of mastitis were found, fed basal total mixed ration (TMR) without supplementation (H); (2) Mastitis cows with milk SCC > 500,000 cells/mL, fed basal TMR without supplementation (M); (3) Mastitis cows fed basal TMR supplemented with 8 g day-1 yeast (M + Y); (4) Mastitis cows fed basal TMR supplemented with 8 g day-1 LAB (M + L); (5) Mastitis cows (milk SCC > 500,000 cells/mL) fed basal TMR supplemented with 4 g day-1 yeast and 4 g day-1 LAB (M + Y + L). Blood and milk sample were collected at day 0, day 20 and day 40. The results showed efficacy of probiotic: On day 20 and day 40, milk SCC in H, M + Y, M + L, M + Y + L was significantly lower than that of M (P < 0.05). Milk concentration of TNF-α, IL-6 and IL-1β in M + Y + L were significantly reduced compared with that of M on day 40 (P < 0.05). Milk Myeloperoxidase (MPO) and N-Acetyl-β-D-Glucosaminidase (NAG) activity of M + Y, M + L, M + L + Y were lower than that of M on day 40 (P < 0.05). At genus level, Staphylococcus, Chryseobacterium and Lactococcus were dominant. Supplementation of LAB decreased abundance of Enterococcus and Streptococcus, identified as mastitis-causing pathogen. The results suggested the potential of LAB to prevent mastitis by relieving mammary gland inflammation and regulating milk microorganisms.

中文翻译:

酵母菌和乳酸菌对奶牛乳腺炎和牛奶菌群组成的影响。

进行该实验以评估酵母和乳酸菌(LAB)对奶牛乳腺炎和牛奶菌群组成的影响。将30头具有相同胎次,泌乳天数的泌乳荷斯坦奶牛随机分为五种处理方法,包括:(1)牛奶SCC <500,000细胞/ mL的健康奶牛,未发现乳腺炎的临床体征,饲喂基础总混合日粮(TMR)不补充(H);(2)乳SCC> 500,000细胞/ mL的乳腺炎奶牛,不添加基础饲喂基础TMR(M);(3)用基础TMR补充8 g day-1酵母菌(M + Y)喂养的乳腺炎奶牛;(4)饲喂基础TMR补充8 g第1天LAB(M + L)的乳腺炎奶牛;(5)饲喂基础TMR补充4 g第1天酵母和4 g第1天LAB(M + Y + L)的乳腺炎母牛(乳SCC> 500,000细胞/ mL)。在第0天收集血液和牛奶样本,第20天和第40天。结果显示了益生菌的功效:在第20天和第40天,H,M + Y,M + L,M + Y + L中的牛奶SCC显着低于M(P <0.05) 。与第40天的M相比,M + Y + L中的牛奶中TNF-α,IL-6和IL-1β的浓度显着降低(P <0.05)。第40天,M + Y,M + L,M + L + Y的牛奶髓过氧化物酶(MPO)和N-乙酰基-β-D-葡萄糖苷酶(NAG)活性均低于M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。在第20天和第40天,H,M + Y,M + L,M + Y + L中的牛奶SCC显着低于M(P <0.05)。与第40天的M相比,M + Y + L中的牛奶中TNF-α,IL-6和IL-1β的浓度显着降低(P <0.05)。第40天,M + Y,M + L,M + L + Y的牛奶髓过氧化物酶(MPO)和N-乙酰基-β-D-葡萄糖苷酶(NAG)活性均低于M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。在第20天和第40天,H,M + Y,M + L,M + Y + L中的牛奶SCC显着低于M(P <0.05)。与第40天的M相比,M + Y + L中的牛奶中TNF-α,IL-6和IL-1β的浓度显着降低(P <0.05)。第40天,M + Y,M + L,M + L + Y的牛奶髓过氧化物酶(MPO)和N-乙酰基-β-D-葡萄糖苷酶(NAG)活性均低于M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。与第40天的M相比,M + Y + L中的牛奶中TNF-α,IL-6和IL-1β的浓度显着降低(P <0.05)。第40天,M + Y,M + L,M + L + Y的牛奶髓过氧化物酶(MPO)和N-乙酰基-β-D-葡萄糖苷酶(NAG)活性均低于M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。与第40天的M相比,M + Y + L中的牛奶中TNF-α,IL-6和IL-1β的浓度显着降低(P <0.05)。第40天,M + Y,M + L,M + L + Y的牛奶髓过氧化物酶(MPO)和N-乙酰基-β-D-葡糖胺酶(NAG)活性均低于M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。M + L + Y低于第40天的M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。M + L + Y低于第40天的M(P <0.05)。在属水平上,葡萄球菌,金黄色葡萄球菌和乳球菌占主导地位。补充乳酸菌会降低肠球菌和链球菌的含量,被确定为引起乳腺炎的病原体。结果表明,乳酸菌具有减轻乳腺炎症和调节乳汁微生物预防乳腺炎的潜力。
更新日期:2020-01-30
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