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Author Correction: Dysfunctional EAT thickness may promote maladaptive heart remodeling in CVD patients through the ST2-IL33 system, directly related to EPAC protein expression.
Scientific Reports ( IF 3.8 ) Pub Date : 2020-01-21 , DOI: 10.1038/s41598-020-58143-y Elena Vianello 1 , Elena Dozio 1 , Francesco Bandera 1, 2 , Gerd Schmitz 3 , Manuela Nebuloni 4 , Erika Longhi 4 , Lorenza Tacchini 2 , Marco Guazzi 1, 2 , Massimiliano Marco Corsi Romanelli 1, 5
Scientific Reports ( IF 3.8 ) Pub Date : 2020-01-21 , DOI: 10.1038/s41598-020-58143-y Elena Vianello 1 , Elena Dozio 1 , Francesco Bandera 1, 2 , Gerd Schmitz 3 , Manuela Nebuloni 4 , Erika Longhi 4 , Lorenza Tacchini 2 , Marco Guazzi 1, 2 , Massimiliano Marco Corsi Romanelli 1, 5
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An amendment to this paper has been published and can be accessed via a link at the top of the paper.
中文翻译：
作者更正：机能失调的EAT厚度可能通过ST2-IL33系统促进CVD患者的适应不良的心脏重塑，这与EPAC蛋白表达直接相关。
该文件的修订版已经发布，可以通过文件顶部的链接进行访问。
更新日期：2020-01-21
中文翻译：
作者更正：机能失调的EAT厚度可能通过ST2-IL33系统促进CVD患者的适应不良的心脏重塑，这与EPAC蛋白表达直接相关。
该文件的修订版已经发布，可以通过文件顶部的链接进行访问。
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