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IL33 contributes to diesel pollution-mediated increase in experimental asthma severity
Allergy ( IF 12.6 ) Pub Date : 2020-01-31 , DOI: 10.1111/all.14181 Eric B Brandt 1 , Paige E Bolcas 1 , Brandy P Ruff 1 , Gurjit K Khurana Hershey 1, 2
Allergy ( IF 12.6 ) Pub Date : 2020-01-31 , DOI: 10.1111/all.14181 Eric B Brandt 1 , Paige E Bolcas 1 , Brandy P Ruff 1 , Gurjit K Khurana Hershey 1, 2
Affiliation
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Exposure to traffic pollution, notably diesel exhaust particles (DEP), increases risk for asthma and asthma exacerbations. The contribution of cytokines generated by stressed lung epithelial cells (IL25, IL33, TSLP) to DEP‐induced asthma severity remains poorly understood.
中文翻译：
IL33 有助于柴油污染介导的实验性哮喘严重程度增加
暴露于交通污染，尤其是柴油机尾气颗粒 (DEP)，会增加哮喘和哮喘恶化的风险。由应激肺上皮细胞（IL25、IL33、TSLP）产生的细胞因子对 DEP 诱导的哮喘严重程度的贡献仍然知之甚少。
更新日期：2020-01-31
中文翻译：
IL33 有助于柴油污染介导的实验性哮喘严重程度增加
暴露于交通污染，尤其是柴油机尾气颗粒 (DEP)，会增加哮喘和哮喘恶化的风险。由应激肺上皮细胞（IL25、IL33、TSLP）产生的细胞因子对 DEP 诱导的哮喘严重程度的贡献仍然知之甚少。
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