Risk for childhood psychopathology is complex and multifactorial, implicating direct and interacting effects of familial and environmental factors. The role of environmental neurotoxicants in psychiatric risk is of growing concern, including polycyclic aromatic hydrocarbons (PAH), common in air pollution. Prenatal PAH exposure is linked to adverse physical, behavioral, and cognitive outcomes as well as increasing psychiatric risk. It is unclear whether environmental exposures, like PAH, magnify the effects of exposure to early life stress (ELS), a critical risk factor for psychopathology. The current work aimed to test potential interactions between prenatal PAH exposure and psychosocial/socioeconomic stress on psychiatric symptoms in school‐age children.

中文翻译：

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产前接触多环芳烃改变了早期生活压力对儿童晚期注意力和思维问题的影响。

儿童精神病理学的风险是复杂的和多因素的，涉及家庭和环境因素的直接和相互作用的影响。环境神经毒物在精神疾病风险中的作用越来越受到关注，包括空气污染中常见的多环芳烃 (PAH)。产前 PAH 暴露与不良的身体、行为和认知结果以及增加的精神病风险有关。目前尚不清楚环境暴露，如 PAH，是否会放大暴露于早期生活压力 (ELS) 的影响，这是心理病理学的一个关键风险因素。目前的工作旨在测试产前 PAH 暴露与学龄儿童精神症状的社会心理/社会经济压力之间的潜在相互作用。