Postharvest physiological deterioration (PPD) of cassava (Manihot esculenta) storage roots is a complex physiological and biochemical process which involve many regulatory networks linked with specific proteins modulation and signaling transduction pathways. However, it is poorly understood regarding biological regulation, and the interactions among protein groups and signals to determine PPD syndrome in cassava storage roots. This review sheds some light on the possible molecular mechanisms involved in reactive oxygen species (ROS), calcium signaling transduction, and programmed cell death (PCD) in cassava PPD syndrome. A model for predicting crosstalk among calcium signaling, ROS and PCD is suggested to fine-tune PPD syndrome. This would clues to cassava molecular breeding to alleviate the PPD effects on the shelf-life.

中文翻译：

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木薯采后生理恶化：一种涉及钙信号、活性氧和程序性细胞死亡的复杂现象。

木薯（Manihot esculenta）贮藏根的采后生理恶化（PPD）是一个复杂的生理和生化过程，涉及许多与特定蛋白质调节和信号转导途径相关的调控网络。然而，人们对木薯贮藏根中的生物调节以及蛋白质组和信号之间的相互作用了解甚少，以确定木薯贮藏根中的 PPD 综合征。这篇综述揭示了木薯 PPD 综合征中活性氧 (ROS)、钙信号转导和程序性细胞死亡 (PCD) 可能涉及的分子机制。建议建立一个预测钙信号、ROS 和 PCD 之间串扰的模型来微调 PPD 综合征。这将为木薯分子育种提供线索，以减轻 PPD 对保质期的影响。