Cigarette smokers exhibit reduced physiological stress reactivity, yet it is unclear whether blunted reactivity predicts differences in subjective recovery and vice versa. The study examined whether basal heart rate and heart rate reactivity were related to recovery in anxiety following stress, and conversely, whether initial self-reported anxiety and anxiety reactivity were related to heart rate recovery. Fifty-six smokers completed a 10-min baseline period, a 4-min stressor, and a 10-min recovery period during which heart rate and anxiety were continuously assessed. Results indicated significant linear (p < .01, d = 0.31) and quadratic (p = .02, d = 0.27) effects of baseline heart rate and reactivity (linear p < .01, d = 0.80; quadratic p < .01, d = 0.66) on recovery in anxiety and significant linear (p < .01, d = 0.88) and quadratic (p < .01, d = 0.74) effects of anxiety reactivity on heart rate recovery. These findings suggest that reduced reactivity in both heart rate and anxiety predicted slower recovery in the opposite domain. Findings offer initial evidence for psychophysiological integration in cigarette smokers.

中文翻译：

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检查吸烟者压力反应性和恢复的生理和心理成分之间的关​​系。

吸烟者表现出降低的生理应激反应性，但是尚不清楚钝化反应性是否预示着主观恢复的差异，反之亦然。该研究调查了基础心率和心率反应性是否与压力后焦虑的恢复有关，反之，最初的自我报告的焦虑和焦虑反应性是否与心率恢复有关。56名吸烟者完成了10分钟的基线期，4分钟的压力源和10分钟的恢复期，在此期间不断评估心率和焦虑。结果表明 基线心率和反应性具有显着的线性（p  <.01，d  = 0.31）和二次（p  = .02，d = 0.27）影响（线性p <.01，d  = 0.80； 焦虑情绪恢复的二次方p  <.01，d = 0.66）和 焦虑反应性对心率恢复的显着线性（p  <.01，d  = 0.88）和二次方（p  <.01，d = 0.74）影响。这些发现表明，心率和焦虑症的反应性降低预示了相反领域的恢复较慢。这些发现为吸烟者的心理生理整合提供了初步的证据。