Placental hypervascularization observed in gestational diabetes mellitus (GDM) may be related to functional availability of vascular endothelial growth factor (VEGF) and its receptors. We aimed to test whether changes in phosphorylation of tyrosine 951 or tyrosine 1175 (pY951 or pY1175) of the vascular endothelial growth factor receptor 2 (VEGFR2) are associated with the proangiogenic state observed in placentas from GDM This article is protected by copyright. All rights reserved.

中文翻译：

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诺丁汉大学第 68 届年会摘要。


妊娠糖尿病 (GDM) 中观察到的胎盘血管过多可能与血管内皮生长因子 (VEGF) 及其受体的功能可用性有关。我们的目的是测试血管内皮生长因子受体 2 (VEGFR2) 酪氨酸 951 或酪氨酸 1175（pY951 或 pY1175）磷酸化的变化是否与 GDM 胎盘中观察到的促血管生成状态相关。本文受版权保护。版权所有。