There are many evidences in support of Behavioral Tagging hypothesis that relies on the setting of a learning tag and the synthesis of plasticity related proteins (PRPs). It explains that how a learning tag produced as a result of weak training can be paired up with PRPs that arrive as a result of novelty and consequently lead to long lasting memories. In the following review we have focused on possible involvement of PKMζ, Arc as PRPs and neurotransmitter receptor systems ACh, metabotropic glutamate in behavioral tagging along with evidences in support of involvement of dopamine, NMDA glutamate and β-adrenergic receptor systems in behavioral tagging.

中文翻译：

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行为标签：PKMζ，弧和神经递质受体系统的作用可能参与

有许多证据支持行为标记假说，该假说依赖于学习标记的设置和可塑性相关蛋白（PRP）的合成。它解释了如何将因训练不足而产生的学习标签与由于新颖性而出现的PRP搭配使用，从而导致持久的记忆。在下面的综述中，我们重点研究了PKMζ，作为PRP的Arc和神经递质受体系统ACh，代谢型谷氨酸可能参与行为标记，以及支持多巴胺，NMDA谷氨酸和β-肾上腺素受体系统参与行为标记的证据。