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TFIID and MYB Share a Therapeutic Handshake in AML.
Cancer Cell ( IF 48.8 ) Pub Date : 2018-01-08 , DOI: 10.1016/j.ccell.2017.12.008 Charles C. Bell , Mark A. Dawson
Cancer Cell ( IF 48.8 ) Pub Date : 2018-01-08 , DOI: 10.1016/j.ccell.2017.12.008 Charles C. Bell , Mark A. Dawson
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Selectively disrupting oncogenic transcription factors in cancer remains an elusive ambition of targeted therapeutics. In this issue of Cancer Cell, Xu et al. provide an elegant proof-of-concept study demonstrating that interaction between MYB and the general transcriptional coactivator TFIID can be specifically disrupted to mediate a therapeutic effect in AML.
更新日期：2018-01-08
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