Cell impurities are an emerging nucleating molecular barriers having the capability in disordering the metabolic chain reactions of proteolysis, glycolysis and lipolysis. Their massive effects induced by copolymer crystal growth in compaction with metal and mineral transients are extended as well as in damaging DNA and mRNA structure motif and other molecular assembly e.g. histones structure unites. Their polycrystalline packing modes, polydispersity and their tendency to surface and interface adhesion prompted us in structuring scaffold biomaterials enriched with biopeptides, layered by phospho-glycerides ester-forms. The interface tension of the formed map is flexible and dependent to the surface exposure and its collapse modes to the surrounding molecular ligands. Thus, the attempts in increasing surface exposure e.g. the viscoelastic of structured lipopeptides and types of formed network structures interplays an extra- conjugating biomolecules having a least cytotoxicity effects to cells constituents. Disulfides molecules are selected to be the key regulatory element in rejoining both lipidic and proteic moieties by disordering atoms status via chemical ionization using organic catalyst. The insertion of methionine based peptidic chain at the lateral surfaces of scaffold biomaterials enhances the electron-meta-static motions by raising a molecular disordering status at distinct regions of the map e.g. epimerization into a nonpolar side that helps the chemical conjunction of disulfide groups with the esterified phosphoglycerides mono-layers. These effects in turn are accomplished by the formation of meso-sphere nonpolar- vesicles. The oxidation of disulfide group would alter the ordering of initial molecules by raising a newly molecular disorders to the map with high polarity to surface regions. In the same time indicates a continuation in the crystallization growth factor via a low chemical lesions between the impurities and a supersaturation in the intra-atomic distances with maximum cross linking to the deformed ligand with scaffold biomaterials.

细胞杂质是一种新兴的成核分子屏障，具有使蛋白水解，糖酵解和脂解作用的代谢链反应紊乱的能力。在金属和矿物瞬态压实过程中，由共聚物晶体生长引起的大量效应得以扩展，并破坏了DNA和mRNA结构基序以及其他分子组装（例如组蛋白结构）。它们的多晶堆积模式，多分散性以及它们在表面和界面上的粘附趋势，促使我们构造出富含生物肽的支架生物材料，所述生物肽由磷酸甘油酯的形式分层。形成的图的界面张力是柔性的，并取决于表面暴露及其对周围分子配体的塌陷模式。因此，增加表面暴露的尝试例如 结构化脂肽的粘弹性和形成的网络结构的类型相互作用，对细胞成分的细胞毒性影响最小的共轭生物分子。选择二硫化物分子作为通过使用有机催化剂通过化学电离使原子状态无序来重新结合脂质和蛋白质部分的关键调节元件。在支架生物材料的侧表面插入基于蛋氨酸的肽链，通过在图谱的不同区域提高分子无序状态（例如差向异构化为非极性面，从而有助于二硫基团与二硫键的化学结合）来增强电子介电运动。酯化的磷酸甘油酯单层。这些作用又通过中球非极性小泡的形成来实现。二硫化物基团的氧化将通过向表面区域以高极性引发新的分子紊乱而改变图谱，从而改变初始分子的顺序。同时表明通过杂质之间的低化学损伤和原子内距离的过饱和，以及通过支架生物材料最大程度地交联到变形配体上的结晶生长因子的延续。