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MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer
Cancer Cell ( IF 48.8 ) Pub Date : 2017-09-11 00:00:00 , DOI: 10.1016/j.ccell.2017.08.008
Surendra K. Shukla , Vinee Purohit , Kamiya Mehla , Venugopal Gunda , Nina V. Chaika , Enza Vernucci , Ryan J. King , Jaime Abrego , Gennifer D. Goode , Aneesha Dasgupta , Alysha L. Illies , Teklab Gebregiworgis , Bingbing Dai , Jithesh J. Augustine , Divya Murthy , Kuldeep S. Attri , Oksana Mashadova , Paul M. Grandgenett , Robert Powers , Quan P. Ly , Audrey J. Lazenby , Jean L. Grem , Fang Yu , José M. Matés , John M. Asara , Jung-whan Kim , Jordan H. Hankins , Colin Weekes , Michael A. Hollingsworth , Natalie J. Serkova , Aaron R. Sasson , Jason B. Fleming , Jennifer M. Oliveto , Costas A. Lyssiotis , Lewis C. Cantley , Lyudmyla Berim , Pankaj K. Singh

(Cancer Cell 32, 71–87; July 10, 2017)

中文翻译:


MUC1和HIF-1alpha信号串扰诱导合成代谢的葡萄糖代谢,从而赋予吉西他滨对胰腺癌的抵抗力。

(癌细胞32,71-87; 2017年7月10日)

更新日期:2017-09-11
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