Rationale: Neutrophils are key effectors in the host's immune response to sepsis. Excessive stimulation or dysregulated neutrophil functions are believed to be responsible for sepsis pathogenesis. However, the mechanisms regulating functional plasticity of neutrophils during sepsis have not been fully determined.

中文翻译：

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自噬引发中性粒细胞在败血症过程中形成中性粒细胞胞外陷阱

基本原理：中性粒细胞是宿主对败血症的免疫反应的关键效应物。过度刺激或嗜中性粒细胞功能失调被认为是脓毒症发病的原因。然而，在败血症期间调节嗜中性粒细胞功能可塑性的机制尚未完全确定。