In states of obesity, the development and fibrotic progression of nonalcoholic fatty liver disease (NAFLD) from nonalcoholic steatohepatitis (NASH) to cirrhosis has been proposed to stem from chronic, low-level type 1 inflammation in adipose tissue. However, new research challenges this view by showing instead that

中文翻译：

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肝：NAFLD免疫发病机制的范式转变

在肥胖状态下，非酒精性脂肪性肝病（NAFLD）从非酒精性脂肪性肝炎（NASH）到肝硬化的发展和纤维化进程已被提议源于脂肪组织中的慢性低水平1型炎症。但是，新的研究挑战性地提出了以下观点：